A neuromuscular condition called myasthenia gravis causes erratic muscle wasting and weakening. It is an autoimmune condition in which the acetylcholine receptors at the post-synaptic neuromuscular junction are blocked by circulating antibodies, preventing the neurotransmitter acetylcholine’s ability to stimulate the muscles. Cholinesterase inhibitors, immunosuppressants, and, in some circumstances, thymectomy are used as medical treatments for myasthenia.

 

Introduction:

 

Chronic myasthenia gravis leads muscles to readily tyre and deteriorate. For instance, if you have myasthenia gravis, you might find that eating makes your jaw muscles weak and fatigued, making it difficult for you to properly chew your food. The muscles that regulate respiration, neck, and limb movements might occasionally also be impacted. Extraocular muscles, which regulate eye movement, as well as muscles that control facial expression, chewing, and swallowing, are the muscles that are most commonly impacted in the early stages of myasthenia gravis. Acute respiratory failure could result from the condition affecting the muscles that regulate breathing (respiration) if it is left untreated.

This particular condition is characterized by the waxing-and-waning weakening of the muscles, which gets worse with use and gets better with rest. Typically, there are times when you may experience greater symptoms, followed by times when they lessen or go away. The nicotinic acetylcholine receptors, which are present in junctions between muscles and the neurological system, are the cause of the erroneous immunological response that results in it. Human antibodies assault acetylcholine receptors, preventing acetylcholine from reaching muscle fibers and leading to weakened muscles.

 

Causes:

 

An autoimmune disease that affects the muscles and nerves is called myasthenia gravis. The body’s immune system incorrectly attacks healthy tissue, leading to autoimmune diseases. Immune cells create antibodies (proteins) that block or harm muscle receptor cells in myasthenia gravis. This stops messages from reaching the muscles from the nerve ends, which causes the muscles to stop contracting (tightening) and weaken. Why certain people’s immune systems develop particular antibodies that obstruct the nerve’s signal to the muscle is not fully known.

 

Symptoms:

 

Myasthenia gravis is characterized by its fatiguability. During moments of exertion, muscles gradually weaken, and they get stronger after resting. Particularly vulnerable are the muscles that manage facial expression, chewing, talking, and swallowing. Additionally impacted are the muscles that manage respiration, neck, and limb motions. The physical examination frequently falls within acceptable ranges. The disorder may develop slowly or suddenly. Symptoms frequently come and go. If the symptoms are vague or fluctuating, the diagnosis of myasthenia gravis may be postponed.

• Signs typically come and go and are inconsistent.
• Symptoms may go away for weeks before returning at a later time.
• In general, weakness gets worse throughout the day and following intense exertion.
• Ptosis, or drooping eyes
• Diaphoresis (diplopia)
• Speech difficulty and slurring
• Having trouble swallowing
• Nasal-like speech
• Muscle weakness in the neck
• Arms and legs are weak.

 

Diagnosis:

 

The diagnosis of myasthenia gravis is frequently overlooked or postponed in those who only have modest weakness or in those whose weakness is limited to a small number of muscles. Examinations of the physical and neurological systems, as well as a study of the patient’s medical history, are required for the first diagnosis of myasthenia gravis. Without any changes in the patient’s capacity for feeling, the doctor searches for impairment of eye movements or muscle weakness. There are a number of tests available to confirm the diagnosis of myasthenia gravis if the doctor has a suspicion.

Acetylcholine receptor antibodies and immunological molecules can both be found using a specialized blood test. The majority of myasthenia gravis patients have very high levels of these antibodies. Recently, it was shown that 30 to 40 percent of people with myasthenia gravis who do not have acetylcholine receptor antibodies also have a second antibody known as the anti-MuSK antibody. Blood can also be examined for the presence of this antibody. Some people with myasthenia gravis, most frequently those with ocular myasthenia gravis, do not have any of these antibodies.

 

Homeopathic Treatment:

 

Homeopathy has proven to be successful in treating myasthenia gravis by reducing the amount of relapses and improving muscle strength and tone. It should be noted, however, that homeopathy may not result in myasthenia gravis disease improvement to the same extent as immunosuppressive drugs. The constitutional approach of homeopathy works more deeply to correct immune system abnormalities, obviating the need for artificial neurotransmitters or steroids as prescribed by traditional treatment. A few homeopathic medicines with features for myasthenia gravis are:

 

Cocculus indicus

• Homeopathic treatment for myasthenia gravis, which causes complete muscle paralysis.
• affects the sensorium first, then the voluntary muscular system.
• Vertigo and disorientation are produced in the sensorium, whereas paralysis and titanic convulsions are produced in the muscular system.
• Muscular weakness caused by paralysis.
• The cervical muscles can’t support the head if they are weak.
• Neck stiffness that hurts to move.
• The hands are drowsy and numb.
• Lower limbs are immobile due to paralysis.
• Sloes go to sleep while sitting with numbness.

Alumina

• Muscular atrophies.
• People lack essential heat and age prematurely as a result of their weakness.
• The symptoms of the remedy include heaviness, stumbling, and sluggishness along with severe constipation.
• When sitting with your legs crossed, your extremities seem paralyzed and your leg especially feels sleepy.
• The soles of the feet are painful and the heels are mushy and swollen.
• Being unable to walk, especially during the day or when the eyes are open.

Curare

• Muscular paralysis caused by myasthenia gravis can be treated with homeopathic medication without affecting consciousness or feeling.
• Muscular paralysis in the lungs.
• The reflex response was lessened
• Spine pain from fatigued.
• Weak and hefty arms. The fingers cannot be raised.
• Pianists’ weakened hands and fingers.
• Legs shake and sway when walking. Paralysis; affliction.

Conium mac

• Conium is a great treatment for symptoms like a stiff, painful gait, shaking, and sudden lack of strength while walking due to myasthenia gravis.
• It is consistent with the debility, hypochondriasis, urinary issues, impaired memory, and sexual debility that are present in this area.
• In bed in the morning, great debility.
• Trembling, palpitations, and a general feeling of weakness.

Gelsemium

• Gelsemium is a homeopathic treatment for myasthenia gravis, which causes numerous muscle groups around the eyes to paralyze. Using homeopathy to treat myasthenia gravesite extremities, larynx, throat, chest, and sphincter.
• Muscle wasting. complete repose and genuflection. a dysfunction in the muscles’ coordination
• A general bowing-down. Trembling, dullness, sleepiness, and vertigo.
• Loss of muscle control power.
• Excessive trembling and limb weakness. hysterical jerks.
• Exhaustion with light exertion.

 

Precautions:

 

• By practicing good hygiene and avoiding sick people, try to prevent infections.
• Quickly treat infections.
• Avoid getting too hot or too cold.
• Prevent overdoing it.
• Discover efficient stress management techniques.